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By R Loch Macdonald; Bryce Weir

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Extra resources for Cerebral vasospasm

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008) incidence of DID, predominantly caused by VSP. 03). 8 NS a From Adams, H. , Kassell, N. , Torner, J. , and Haley, E. C. (1987). Predicting cerebral ischemia after aneurysmal subarachnoid hemorrhage: Influences of clinical condition, CT results, and antifibrinolytic therapy. Neurology 37, 1586-1591. b NS, not significant. 05). The risk seemed to be greatest for patients who were admitted with normal systolic blood pressure. Perhaps the antifibrinolytics increased the dose of vasoconstrictor agonist by preserving the clot and the antihypertensive blunted the homeostatic hypertensive response that would have countered the angiographically evident VSP (45).

Kassell, N. , Torner, J. , and Haley, E. C. (1987). Predicting cerebral ischemia after aneurysmal subarachnoid hemorrhage: Influences of clinical condition, CT results, and antifibrinolytic therapy. Neurology 37, 1586-1591. b NS, not significant. 05). The risk seemed to be greatest for patients who were admitted with normal systolic blood pressure. Perhaps the antifibrinolytics increased the dose of vasoconstrictor agonist by preserving the clot and the antihypertensive blunted the homeostatic hypertensive response that would have countered the angiographically evident VSP (45).

A close correlation between the clinical grade based on GCS and the incidence of VSP diagnosed either clinically or by CT evidence of infarction was evident. In this Japanese series, VSP was more common in the older age group. The site of aneurysm or the day of surgery had no apparent influence on the incidence of VSP or its outcome. Poor outcome attributable to VSP occurred in 1 2 ~ which was one-third of the cases of poor outcome from all causes. 1 days post-SAH. 4 days post-SAH (range, 5-12).

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