By R.W. Baumgartner, J. Bogousslavsky, V. Caso, M. Paciaroni
Major foreign specialists summarize updated findings at the presentation, analysis, pathogenesis and remedy of cerebral artery dissection. a number of issues, resembling the 1st animal version of cervical artery dissection, the epidemiology and its organization with connective tissue abnormalities in dermis and arteries, genetic techniques within the learn of probability elements in addition to the most etiologies of spontaneous and demanding cervical artery dissection are mentioned. The e-book additionally provides an perception into the scientific manifestations of carotid and vertebral artery dissection and to the probabilities and obstacles of the most diagnostic instruments, ultrasound, CT and MR imaging, and angiography. The final a part of the e-book offers the diagnosis, thrombolysis and antithrombotic treatment of cervical artery dissection and concludes with an outline of intracranical dissection. experts in neurology, neuroradiology, neurosurgery, inner drugs in addition to common practitioners will take advantage of this publication.
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Additional resources for Handbook on Cerebral Artery Dissection
Am J Physiol 1986;250:H1145–H1149. Palmer RM, Ferrige AG, Moncada S: Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor. Nature 1987;327:524–526. Pohl U, Herlan K, Huang A, Bassenge E: EDRF-mediated shear-induced dilation opposes myogenic vasoconstriction in small rabbit arteries. Am J Physiol 1991;261:H2016–H2023. Celermajer DS, Sorensen KE, Gooch VM, Spiegelhalter DJ, Miller OI, Sullivan ID, Lloyd JK, Deanfield JE: Non-invasive detection of endothelial dysfunction in children and adults at risk of atherosclerosis.
Basel, Karger, 2005, vol 20, pp 54–63 Traumatic Cervical Artery Dissection Krassen Nedeltcheva, Ralf W. Baumgartnerb a Institute of Diagnostic and Interventional Neuroradiology, University Hospital of Bern, Bern, and bDepartment of Neurology, University Hospital of Zürich, Zürich, Switzerland Abstract Traumatic cervical artery dissection (TCAD) is a complication of severe blunt head or neck trauma, the main cause being motor vehicle accidents. 53% for traumatic vertebral artery dissections (TVAD) among blunt trauma victims are reported.
However, Willis et al.  described several patients with unstable cervical fractures affecting also the transversal foramina, who presented in 46% asymptomatic TVAD. Pathogenesis of TCAD In 1974, Crissey and Bernstein  described four mechanisms leading to injury of the cervical ICA: (1) direct blow to the neck, (2) neck hyperextension associated with rotation, (3) blunt intraoral trauma, and (4) basilar skull fracture involving the carotid canal. Direct blow to the neck was responsible for up to 50% of cases in this report.